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Description

J Dev Biol

Advanced glycation end-products activate the renin-angiotensin system through the RAGE/PI3-K signaling pathway in podocytes

In hosts where the reduction of thioredoxin and glutathione is impaired by mutations to the thioredoxin reductases and glutathione reductase genes, the resultant oxidizing conditions allow the formation of disulfide bonds in expressed proteins located in bacterial cytoplasm (Bessette et al., 1999)