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Animal toxicology signals Two animal findings reviewed by FDA are worth tracking even if they did not surface clinically

(h)GSH Depletion Negatively Influences Nodule Development, While Exogenous Glutathione Application Promotes Infection and Defense Responses in sym33-3 Mutants and the Maturation of Infected Cells in sym40-1 Mutants The expression levels of GSH1 , GSHS , Cyp15a, TPP , PR1 , and PR10 were upregulated in (h)GSH-depleted wild-type roots with nodules (Figure 7)

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Limited role for CXC chemokines in the pathogenesis of alpha-naphthylisothiocyanate-induced liver injury

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