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The apo(a) component increases atherosclerosis and promotes clotting by interfering with the bodys clot-dissolving mechanisms

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Under cold conditions, FGF21 derived from BAT serves as a mediator of 3-adrenergic-dependent GDF15 gene transcription and is released in BAT, which suppresses proinflammatory gene expression and decreases TNF and CCL2 secretion in macrophages

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The potential anti-inflammatory mechanisms of SHFKO may be related to multiple bioactive components identified in our analysis: phellodendrine has been associated with reduced IL-6 and TNF- transcription in studies involving the IL-17/NF-B signaling axis

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