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From a therapeutic perspective, anti-inflammatory drugs may partially restore neurogenesis and represent an avenue for adjunct therapy in ameliorating neuronal decline [110]

Evidence is primarily derived from small clinical and preclinical studies

5HT receptor activation alleviates neuropathic painrelated antidepressant via the ACcAMPPKAMEKCREBBDNF cascade [26], and oxytocin protects dopaminergic function against corticosterone through PKA/CREB signaling [27]

Senolytic compounds may help eliminate these cells before they become a problem

According to the study of Zhang, lncRNA FLVCR1-AS1 sponges miR-513c and increases c-Met expression in HCC cells, which induces HCC progression (Zhang et al., 2018a)