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Erythrocyte glutathione deficiency in symptom-free HIV infection is associated with decreased synthesis rate Publication: American Journal of Physiology-Endocrinology and Metabolism Volume 276, Issue 1 Abstract Although several studies have documented intra- and extracellular glutathione (GSH) deficiency in asymptomatic human immunodeficiency virus (HIV) infection, the mechanisms responsible for the altered GSH homeostasis remain unknown

Our current understanding of the aging process positions mTORC1 simultaneously as a gate-keeper of cellular physiology and as the main driver of most age-associated pathological alterations