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Studies indicate that FOS enhances the phagocytic and bactericidal capacity of phagocytes (neutrophils and macrophages) against invading pathogens, including promoting phagocytosis, inducing reactive oxygen species (ROS) generation, and stimulating the production of extracellular traps (ETs) (Figure 1B

Basal mitophagy occurs independently of PINK1 in mouse tissues of high metabolic demand

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Influenza virus infection activates TAK1 to suppress RIPK3-independent apoptosis and RIPK1-dependent necroptosis