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In general, the BBB can be regarded as an organ that serves to protect the homeostasis of the brain

Transitions are biomarker-gated: move from acute subacute when damage markers fall and signalling proxies (sulfenylation/Prx cycling) re-emerge, progress to chronic when redox buffers and function normalize

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And that is not observed: Children with IGF1 defects suffer from far more severe fetal growth retardation with birth weight SDS around -4 and birth length SDS of -5 to -6 (reviewed in (58)