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Now, as for the reversibility of nerve damage: In terms of neurological recovery from B12 deficiency, studies conducted on rats so far indicate that methyl B12 may promote the regeneration of nerves: Schwann cells (SCs) are constituents of the peripheral nervous system

No significant changes or trends were observed in any treatment group with the Oral Glucose Tolerance Test (OGTT)

This hormone slows gastric emptying, increases insulin secretion, and suppresses appetite through both peripheral and central mechanisms

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The darkening of sole and palmar creases, interphalangeal joints, and terminal phalanges was also observed [3,4,5]

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