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This response was caused primarily by GH hypersecretion, as expression of a GH antagonist resulted in improvement of glucose homeostasis [129, 229]

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This hypothesis has been documented for many drugs, as indicated by the Target Concentrations and Toxic Concentrations columns in Table 31

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in 2017, it has been the subject of approximately 15-20 peer-reviewed publications spanning metabolic disease, muscle biology, cardiovascular research, neurodegenerative disease, and cancer metabolism