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Glycolysis controls the induction of human regulatory T cells by modulating the expression of FOXP3 exon 2 splicing variants

and subsequently to activate the thiol group of GSH, enabling the nucleophilic attack upon the substrate

This pathway, mediated by the LOX-1 receptor, connects elevated cholesterol levels to enhanced NO production, fostering a tumor-permissive microenvironment

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Using roGFP-iL, we were able to determine that the redox potential imposed onto a thiol pair in the periplasm is -229 mV

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