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This, in turn, limits the availability of SAMe for methylation 12

In fact, a study using CNS samples from EAE demonstrated that microglia with differential expression of myeloid activation marker (MHC classII, CD86+), in addition to TREM2+, progressively increase from the asymptomatic phase to the peak of EAE (144)

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Sample sizes were chosen based on existing literature in the field for similar experiments 39,43

Although meaningful results have been achieved in understanding the pathogenesis and therapeutic targets of UC, progress in the treatment of UC remains slow