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doi: 10.1007/s10637-021-01111-8 208 FangYLiuWTangZJiXZhouYSongSet al

This delineates a preference of the PINK1/Parkin pathway for targeting and eliminating dysfunctional mitochondria, whereas the receptor pathway primarily addresses excess mitochondria [52, 53]

Particularly, ferroptosis intersects with characteristic diabetic mechanisms, including oxidative stress, lipid peroxidation, and mitochondrial dysfunction